By F. Z. Meerson (auth.), E. Chazov M.D., V. Saks M.D., G. Rona M.D. (eds.)

This quantity of Advances in Myocardiology is derived from part of the lawsuits of the tenth Congress of the overseas Society for center study, which was once held in Moscow on September 23-29, 1980. This booklet comprises chosen papers that have been prepared in sections, Cardiac Hypertrophy, model, and Pathophysiology and Cardiac Hypoxia, Is­ chemia, and Infarction. the 1st part, at the pathophysiology of middle hypertrophy and failure, includes 24 chapters that target the derangement of biochemical, physiological, and immunological techniques through the de­ velopment of middle ailment because of a large choice of pathogenic elements. many of the fresh advancements in knowing the myocardial man made ma­ chinery in center hypertrophy also are defined in those papers, and we think that the contents of this part will stimulate additional examine within the sector of center ailment. the second one part, which quite often bargains with myocardial ischemia, includes 35 chapters offering the required again­ flooring for the prognosis of ischemic middle sickness and a few attainable ther­ apeutic methods. It not often should be emphasised that ischemic middle affliction is an incredible reason behind demise in hugely industrialized nations, yet regrettably the precise mechanism wherein ischemic insult results in the advance of center telephone harm is way from understood. we're hopeful that the articles during this part will supply useful info during this box and therefore might help in enhancing the remedy of ischemic center disease.

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Propranolol, 10- 5 M, was added 10 min before the epinephrine in some experiments. In some experiments (propranolol plus epinephrine), Metabolism in Catecholamine Injury 25 the heart rate was kept at 360 beats/min by means of atrial pacing with an electronic stimulator (Grass Model S88, 5-V square wave, duration 4 msec). Mechanical Performance Cardiac output was the sum of the aortic output and coronary flow. Heart rate was taken from the ECG recorded from the aortic and atrial perfusion cannulae.

See Figure I. f See discussion in text. I. Control value 2. Control % change at 35 min 3. Epinephrine 10- 8 M 4. Epinephrine 10- 7 M 4. Epinephrine 10- 6 M 6. Theophylline 10- 3 M 7. Dibutyryl cAMP 10- 4 M 8. mol CJg) (nmollg) Table 4. Comparative Effects of Epinephrine and Dibutyryl Cyclic AMP on Mechanical and Metabolic Parameters of Isolated Perfused Working Rat Hearta f. \0 .... E. :1 ..... ~. ~ :r n ~ () 5· 3 s:: <> 40 A. R. Horak. and L. H. Opie Epinephrine versus Theophylline versus Dibutyryl Cyclic AMP These agents had dissimilar effects on the heart.

Specific inhibitors and promoters of calcium action in the excitation-contraction coupling of heart muscle and their role in the prevention or production of myocardial lesions. In: P. Harris and L. H. ), Calcium and the Heart, pp. 135-188. Academic Press, London, New York. 7. , Doring, H. , and Leder, O. 1974. Myocardial fiber necrosis 42 8. 9. 10. II. 12. 13. 14. 15. 16. 17. 18. 19. 20. 21. 22. 23. 24. 25. A. R. Horak and L. H. Opie due to intracellular Ca overload-a new principle in cardiac pathophysiology.

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